Wikipedia says that “grellin is a hormone produced mainly by P/D1 cells lining the fundus of the human stomach and epsilon cells of the pancreas that stimulates hunger. Ghrelin levels increase before meals and decrease after meals. It is considered the counterpart of the hormone leptin, produced by adipose tissue, which induces satiation when present at higher levels. In some bariatric procedures, the level of ghrelin is reduced in patients, thus causing satiation before it would normally occur.”
I heard Dr. Oz on his recent tv show mention that hormone and I went to look it up. Editors of a study on ghrelin quoted by Wikipedia, clarify that rapid emptying of the stomach is closely related to overeating and obesity and that gastric distension acts as a satiety signal. But corsets prohibit stomach distension. Does corseting substitute for gastric distension and also delay emptying of the stomach so that the wearer feels full faster?
The above brief introduction by Wikipedia doesn’t give me the answer to a few hunger-related questions I failed to address in my waist training book simply because I don’t have the answer yet. Those questions relate not only to what causes hunger and what’s related to it, but to why squeezing the stomach with a corset, aside from physically reducing the area one can stuff with food, reduces hunger not just in the moment–but permanently disappears hunger in a matter of days. That’s something I observe personally, and something that is consistently reported to me by other corset enthusiasts and by many of the students whom I coach in waist training. If ghrelin levels increase before meals yet a corseted person doesn’t feel hungry, what is happening?
As with any medical issue, there is a level of complexity that a layperson cannot possibly comprehend, and research changes the horizon study-to-study. It’s a topic I’ll pursue in the near future, but I’d like to hear about your practical hunger experiences of all kinds, especially with consistent corseting.